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Hemodynamic and autonomic nervous system (ANS) output changes after bariatric surgery.
Session:
Posters - P. Other
Speaker:
Lígia Lopes Mendes
Congress:
CPC 2021
Topic:
P. Other
Theme:
37. Miscellanea
Subtheme:
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Session Type:
Posters
FP Number:
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Authors:
Lígia Mendes; Isabel Melo; Raquel Seiça; Hans Heickoff
Abstract
<p style="text-align:start"><span style="font-size:medium"><span style="font-family:Cambria"><span style="color:#000000"><strong>Background:</strong>Obesity is a well-recognized cardiovascular risk factor. However, whether its effects are mediated by overload and hemodynamic strain and/or autonomic nervous system (ANS) imbalance, is not yet completely understood. </span></span></span></p> <p style="text-align:start"><span style="font-size:medium"><span style="font-family:Cambria"><span style="color:#000000"><strong>Aims:</strong><span style="font-size:13pt"><span style="font-family:Times"><span style="color:#1a1718">The purpose of the present study is to investigate the effects of laparoscopic sleeve gastrectomy (LSG) on arterial strain/stiffness and variables dependent on ANS imbalance in subjects with severe or morbid obesity.</span></span></span></span></span></span></p> <p style="text-align:start"><span style="font-size:medium"><span style="font-family:Cambria"><span style="color:#000000"><strong>Methods:</strong><span style="font-size:13pt"><span style="font-family:Times"><span style="color:#1a1718">Fourteen female patients undergoing LSG for standard indications were enrolled. All subjects underwent a physical examination with biometric evaluation, pulsed Doppler to assess carotid-femoral pulse wave velocity (PWV), and 24h Holter to measure mean heart rate and SDNN (standard deviation normal-to-normal intervals), before surgery (0M) and 6 months post-operatively (6M). </span></span></span><span style="font-size:13pt"><span style="font-family:Times">Values are presented as the mean with standard deviation. Pre-and postoperative time points were compared using a paired t-test. </span></span></span></span></span></p> <p style="text-align:start"><span style="font-size:medium"><span style="font-family:Cambria"><span style="color:#000000"><strong>Results:</strong>The mean weight at 0M was 103.5 ±11.5 Kg and 74.5 ±9 kg at 6M (p< 0.0001), fat mass 45 ±5.7% before and 33.5 ±7.8 % at 6M. Arterial strain/stiffness and ANS dependent variables are presented in table 1.</span></span></span></p> <p style="text-align:start"> </p> <p style="text-align:start"><span style="font-size:medium"><span style="font-family:Cambria"><span style="color:#000000">Weight loss at 6M only had correlation with the systolic blood pressure (r=0.58, p=0,029).</span></span></span></p> <p style="text-align:start"> </p> <p style="text-align:start"><span style="font-size:medium"><span style="font-family:Cambria"><span style="color:#000000"><strong>Conclusions:</strong>After LSG, the changes in systolic and diastolic blood pressure as well as in leg-arm index suggest a correction in the amount of strain imposed in arterial pool, and the increment of the SDNN indicate an increase of vago-sympathetic balance. Both improvements that cannot be exclusively attributed to the weight loss.</span></span></span></p> <p style="text-align:start"> </p>
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