Login
Search
Search
0 Dates
2024
2023
2022
2021
2020
2019
2018
0 Events
CPC 2018
CPC 2019
Curso de Atualização em Medicina Cardiovascular 2019
Reunião Anual Conjunta dos Grupos de Estudo de Cirurgia Cardíaca, Doenças Valvulares e Ecocardiografia da SPC
CPC 2020
CPC 2021
CPC 2022
CPC 2023
CPC 2024
0 Topics
A. Basics
B. Imaging
C. Arrhythmias and Device Therapy
D. Heart Failure
E. Coronary Artery Disease, Acute Coronary Syndromes, Acute Cardiac Care
F. Valvular, Myocardial, Pericardial, Pulmonary, Congenital Heart Disease
G. Aortic Disease, Peripheral Vascular Disease, Stroke
H. Interventional Cardiology and Cardiovascular Surgery
I. Hypertension
J. Preventive Cardiology
K. Cardiovascular Disease In Special Populations
L. Cardiovascular Pharmacology
M. Cardiovascular Nursing
N. E-Cardiology / Digital Health, Public Health, Health Economics, Research Methodology
O. Basic Science
P. Other
0 Themes
01. History of Cardiology
02. Clinical Skills
03. Imaging
04. Arrhythmias, General
05. Atrial Fibrillation
06. Supraventricular Tachycardia (non-AF)
07. Syncope and Bradycardia
08. Ventricular Arrhythmias and Sudden Cardiac Death (SCD)
09. Device Therapy
10. Chronic Heart Failure
11. Acute Heart Failure
12. Coronary Artery Disease (Chronic)
13. Acute Coronary Syndromes
14. Acute Cardiac Care
15. Valvular Heart Disease
16. Infective Endocarditis
17. Myocardial Disease
18. Pericardial Disease
19. Tumors of the Heart
20. Congenital Heart Disease and Pediatric Cardiology
21. Pulmonary Circulation, Pulmonary Embolism, Right Heart Failure
22. Aortic Disease
23. Peripheral Vascular and Cerebrovascular Disease
24. Stroke
25. Interventional Cardiology
26. Cardiovascular Surgery
27. Hypertension
28. Risk Factors and Prevention
29. Rehabilitation and Sports Cardiology
30. Cardiovascular Disease in Special Populations
31. Pharmacology and Pharmacotherapy
32. Cardiovascular Nursing
33. e-Cardiology / Digital Health
34. Public Health and Health Economics
35. Research Methodology
36. Basic Science
37. Miscellanea
0 Resources
Abstract
Slides
Vídeo
Report
CLEAR FILTERS
Left atrial and ventricular function in hypertrophic cardiomyopathy assessed by speckle tracking in transthoracic echocardiography
Session:
Posters 2 - Écran 09 - Miocárdio e Pericárdio
Speaker:
Silvia Aguiar
Congress:
CPC 2018
Topic:
F. Valvular, Myocardial, Pericardial, Pulmonary, Congenital Heart Disease
Theme:
17. Myocardial Disease
Subtheme:
17.3 Myocardial Disease – Diagnostic Methods
Session Type:
Posters
FP Number:
---
Authors:
Sílvia Aguiar Rosa; Luisa Moura Branco; Ana Galrinho; Joana Gomes Feliciano; António Fiarresga; Ana Teresa Timóteo; Joao Abreu; Ana Abreu; Pedro Rio; Guilherme Portugal; André Viveiros Monteiro; Pedro Pinto Teixeira; Rita Ilhão Moreira; Madalena Coutinho Cruz; Pedro Daniel; Dra. Inês Rodrigues; Rui Cruz Ferreira
Abstract
<p><strong>Introduction: </strong>Transthoracic Doppler echocardiography (TTE) is the first line imaging method to evaluate hypertrophic cardiomyopathy (HCM) patients (P). The aim is to assess left atrial (LA) and ventricular (LV) strain by TTE in HCM.</p> <p><strong>Methods:</strong> Retrospective analysis of P with normal left ventricular ejection fraction who underwent a comprehensive TTE for HCM evaluation, in a tertiary centre. LA function was assessed by determination of longitudinal strain (LS) and strain rate (SR) in apical 4 chambers view in 3 phases: reservoir, conduit and pump booster. LV global longitudinal strain (GLS) was assessed in apical 4, 2 and 3 chambers view. Radial strain and strain rate was assessed at papillary muscles level in short axis view.</p> <p><strong>Results: </strong>52 P were enrolled, mean age 66.5±15.9 years, 44% males, 38% with obstructive HCM.</p> <p>LA LS in conduit and pump booster phases were worse in P with LA dilatation (>40mm) (-0.43±037 vs -0.88±0.64 s<sup>-1</sup>, p=0.019 and -0.74±0.62 vs -1.08±0.27 s<sup>-1</sup>, p=0.037, respectively).</p> <p>LA LS was substantially different in sinus rhythm and atrial fibrillation P (LS in conduit phase: 8.72±6.56 vs 0.52±2.53%, p=0.006; LS in reservoir phase: 13.29±8.17 vs 6.47±2.72%, p=0.036), as well as SR in reservoir phase (0.63±0.57 vs 0.38±0.25 s<sup>-1</sup>, p=0.048).</p> <p>LA strain rate in conduit phase was significantly different in obstructive and non-obstructive HCM (-0.31±0.44 vs -0.65±0.42 s<sup>-1</sup>, p=0.002) suggesting a more relevant impairment in diastole, particularly in rapid filling phase, in obstructive HCM P.</p> <p>In overall population, LV GLS was -13.92±6.82%, interventricular septum (IVS) peak longitudinal strain -7.53±6.82% and IVS peak radial strain 22.88±16.64%. There was not a significant difference in these parameters between obstructive and non-obstructive HCM.</p> <p>There was a concordance between tissue Doppler (septal s’) and IVS peak radial strain. P with septal s’ >6 presented IVS peak radial strain of -12.67±10.67% contrasting with -5.84±5.08% in P with lower s’ (p=0.010).</p> <p>IVS radial strain rate was higher in P with LA dilatation (2.36±1.13 vs 1.50±0.50 s<sup>-1</sup>, p=0.029).</p> <p>P with impairment in LV function assessed by GLS (> -18%) presented lower LA SR in reservoir phase (0.56±0.49 vs 0.93±0.31s<sup>-1</sup>, p=0.010) and in pump booster phase (-0.70±0.54 vs -8.07±20.6s<sup>-1</sup>, p=0.016).</p> <p><strong>Conclusion: </strong>Changes in LA strain and strain rate were more evident in the presence of LA dilatation, atrial fibrillation and obstructive HCM. Even with a normal LV ejection fraction, HCM P presented an impairment in GLS, IVS peak longitudinal and radial strain. Lower Tissue Doppler velocity in IVS was associated with worse radial strain in this segment. There was a concordance between the impairment of LA and LV function assessed by strain study.</p> <p> </p>
Slides
Our mission: To reduce the burden of cardiovascular disease
Visit our site